Which PCOS / PMOS phenotype do you have?
The Rotterdam criteria (2003) define four phenotypes: A, B, C and D. Your phenotype influences your metabolic prognosis and treatment approaches. This mini-quiz, based on the ESHRE/Monash 2023 international guideline, gives you an orientation — it does not replace medical advice.
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Do you have excessive body hair (face, chin, jaw, chest, abdomen, back, buttocks)?
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The 4 phenotypes in detail
Rotterdam phenotypes are based on the combination of three criteria: (1) clinical or biochemical hyperandrogenism, (2) ovulatory dysfunction, (3) polycystic ovarian morphology or elevated AMH.
Phenotype A — full
All three criteria are present. This is the most metabolically severe phenotype, with increased risk of insulin resistance, type 2 diabetes and metabolic syndrome.
Phenotype B — hyperandrogenism + ovulatory dysfunction
Without polycystic morphology on ultrasound. High cardiometabolic risk, close to phenotype A.
Phenotype C — ovulatory
Hyperandrogenism + polycystic morphology with broadly regular cycles. Spontaneous fertility may be preserved. Skin symptoms often dominate.
Phenotype D — non-hyperandrogenic
Ovulatory dysfunction + polycystic morphology without hyperandrogenism. The least metabolically severe phenotype, but still requires monitoring.
Why phenotype changes management
Phenotype guides priorities. Phenotype A calls for close cardiometabolic monitoring (HbA1c, lipids, blood pressure), while phenotype C tends to focus more on dermatological management (acne, hirsutism, alopecia). This stratification is one reason the Endocrine Society and Monash University renamed the syndrome to PMOS in May 2026 — to acknowledge it is not just an ovarian problem.
General information. This quiz is based on the Rotterdam criteria (2003) and the ESHRE/Monash 2023 update. It does not provide a diagnosis. Sources: scientific sources.